Oxygen
Iron supports hemoglobin and oxygen delivery to tissues.
LIV is a daily iron drink powder designed for women, combining 18 mg elemental iron, vitamin C, key blood and energy cofactors, and a gut-comfort focused support blend.
LIV is a daily iron drink powder designed for women, one scoop mixed into water. It’s built around 18 mg elemental iron per serving (from ferrous bisglycinate) and paired with vitamin C (from acerola) plus selected cofactors involved in blood and energy metabolism (B2, B6, folate, B12, vitamin D3, vitamin A, copper). It also includes a gut-supporting “comfort core”: acacia fiber (prebiotic), a probiotic strain (Lactobacillus rhamnosus), and ginger extract, chosen to support day to day tolerance.
The goal is simple: daily iron support that women can actually take consistently, designed to be gentler on the stomach (less nausea, constipation, and gut discomfort than many traditional iron pills), optimized for absorption, and built around nutrients women commonly need alongside iron (Tolkien et al., 2015; Perrar et al., 2023).
Iron is essential for:
Iron depletion is common in women due to menstrual blood loss, pregnancy-related needs, and dietary factors. Importantly, iron depletion can exist before anemia is diagnosed. So “normal hemoglobin” doesn’t always mean “optimal iron status” (Zimmermann & Hurrell, 2007). Finnish population data, for example, has shown meaningful rates of low ferritin and iron depletion in women of reproductive age (Haltia et al., 2003).
Iron supports hemoglobin and oxygen delivery to tissues.
Iron participates in cellular energy metabolism.
Adequate iron status can influence recovery, performance, and cognition.
LIV is formulated around three real-world reasons iron routines fail: absorption, utilization, and adherence.
1) Absorption: getting non-heme iron across the gut wall
Most supplements deliver non-heme iron, which is sensitive to meal context and absorption inhibitors (like phytates and polyphenols). Two evidence based levers matter here:
2) Utilization: supporting red blood cell production and iron handling
Iron doesn’t act alone. Several nutrients support pathways that determine how effectively the body uses iron:
LIV includes these as part of a “systems” approach: not just iron in isolation, but iron plus key cofactors that support the same physiological endpoint, healthy oxygen delivery and energy metabolism.
3) Adherence: reducing the “I quit iron” problem (gut, stomach, constipation)
One of the most consistent barriers to successful iron supplementation is gastrointestinal side effects. A large systematic review and meta-analysis found ferrous sulfate (a common “standard” iron pill) significantly increases GI side effects compared with placebo (Tolkien et al., 2015).
LIV’s central iron form is ferrous bisglycinate, an amino-acid chelate often selected for better tolerability. A 2023 systematic review and meta-analysis of randomized trials found ferrous bisglycinate was associated with fewer reported GI adverse events (notably in pregnancy trials) and favorable hematologic outcomes in that population (Perrar et al., 2023). Better tolerability supports the real goal: consistency, which is what ultimately supports changes in iron status over time.
LIV’s gut-support ingredients are there for a practical reason: iron routines often fail in the gut.
LIV is designed for women who want daily iron support without the typical downsides, especially those who:
Many iron products ask women to choose between absorption and tolerability and then rely on willpower to maintain a routine that often causes discomfort.
LIV is different because it’s built as a system:
Cherbut, C., Michel, C., Raison, V., Kravtchenko, T., & Severine, M. (2003). Acacia gum is a bifidogenic dietary fibre with high digestive tolerance in healthy humans. Microbial Ecology in Health and Disease, 15(1), 43–50.
EFSA Panel on Dietetic Products, Nutrition and Allergies (NDA). (2016). Lactobacillus plantarum 299v and an increase of non-haem iron absorption: Evaluation of a health claim pursuant to Article 13(5) of Regulation (EC) No 1924/2006. EFSA Journal, 14(6), 4550.
Ernst, E., & Pittler, M. H. (2000). Efficacy of ginger for nausea and vomiting: A systematic review of randomized clinical trials. British Journal of Anaesthesia, 84(3), 367–371.
Hallberg, L., Brune, M., & Rossander, L. (1986). Effect of ascorbic acid on iron absorption from different types of meals. Human Nutrition. Applied Nutrition, 40(2), 97–113. (PMID: 3700141)
Hallberg, L., Brune, M., & Rossander, L. (1991). Ascorbic acid prevents the dose-dependent inhibitory effects of polyphenols and phytates on nonheme-iron absorption. The American Journal of Clinical Nutrition, 53(2), 537–541.
Haltia, P., et al. (2003). Iron status of adults in the capital area of Finland. European Journal of Nutrition. (PMID: 14564462)
Hoppe, M., Önning, G., Hulthén, L., & Berggren, A. (2015). Probiotic strain Lactobacillus plantarum 299v increases iron absorption from an iron-supplemented fruit drink: A double-isotope cross-over single-blind study in women of reproductive age. British Journal of Nutrition, 114(8), 1195–1202.
Hurrell, R., & Egli, I. (2010). Iron bioavailability and dietary reference values. The American Journal of Clinical Nutrition, 91(5), 1461S–1467S.
Marx, W., et al. (2020). Ginger on human health: A comprehensive systematic review of 109 randomized controlled trials. Nutrients, 12(1), 157.
Perrar, I., et al. (2023). The effects of oral ferrous bisglycinate supplementation on hemoglobin and ferritin concentrations in adults and children: A systematic review and meta-analysis of randomized controlled trials. Nutrition Reviews. (PMID: 36728680)
Tolkien, Z., Stecher, L., Mander, A. P., Pereira, D. I. A., & Powell, J. J. (2015). Ferrous sulfate supplementation causes significant gastrointestinal side-effects in adults: A systematic review and meta-analysis. PLOS ONE, 10(2), e0117383.
Zahra, A., et al. (2021). The effect of gum Arabic (Acacia senegal) on cardiovascular risk factors and gastrointestinal symptoms in adults at risk of metabolic syndrome: A randomized clinical trial. Nutrients, 13(1), 194.
Zimmermann, M. B., & Hurrell, R. F. (2007). Nutritional iron deficiency. The Lancet, 370(9586), 511–520.